Hiển thị các bài đăng có nhãn Brain Damage. Hiển thị tất cả bài đăng
Hiển thị các bài đăng có nhãn Brain Damage. Hiển thị tất cả bài đăng

Thứ Tư, 9 tháng 11, 2016

The Neurotoxicity Of Vaccines

By: Alexandria Addesso

Illness is something no one wants to suffer from. Over-the-counter, prescription drugs, and holistic treatments are vigorously sought after to alleviate the slightest form of suffering. In the past hundred years vaccinations have become commonplace when trying to combat both deadly diseases and common ailments. Yet there has been a movement against vaccinations springing up in the last two decades.

Of all vaccinations the flu shot has become the most common and the most frequently administered vaccine being that people usually get one annually at the onset of the flu season. It is well known that if a person already has a compromised immune system at the time of receiving the shot they are highly likely to become very ill. Another controversial and alarming aspect about the flu shot is the high amounts of mercury it contains. Recent lab tests conducted by the Natural News Forensic Food Lab found that seasonal flu vaccines contain 25 thousand times more mercury than is legally allowed in drinking water. Mercury is one of the most toxic metals, it is also particularly neurotoxic. Mercury is known to penetrate and damage the blood-brain barrier very rapidly, leading to a dysfunction of the blood-brain barrier system. Women who are pregnant are usually told to abstain from seafood due to it containing mercury, yet they are encouraged to receive a flu shot as well as other high-risk groups such as children and the elderly.



The flu shot is not the only vaccination known to be neurotoxic, nearly all the vaccines we often push on newborns and infants are. For quite some time autism has been seen to be directly linked to newborn and infantile vaccinations, yet many try to argue the opposite despite autism being almost completely unheard of before mass vaccination programs were initiated in Western nations.

"Dyslexia, minimal brain damage, ADD, autism, allergies, visual and many other neurologic diseases grouped together as ‘developmental disabilities, ’barely existed before mass vaccination programs,” said Dr. Ted Koren. “Probably twenty percent of American children, one youngster in five-suffers from, a 'developmental disability.' This is a stupefying figure developmental disabilities are nearly always generated by encephalitis. And the primary cause of encephalitis in the United States and other industrialized countries is the childhood vaccination program.”

What is most alarming about the childhood vaccination program is that it is a major requirement for public schooling, only sometimes avoided by citing religious belief. A quick Google search can uncover a stories of parents telling stories of how their babies had febrile seizure right after inoculations, some numerous times. Yet their first-hand accounts, either go unheard or purposely silenced. Their previously healthy babies having life-long neurological disorders afterwards.



As people we cannot submit to the state’s demands simply because we are threatened. The youth or other high-risk groups can no longer be a sacrificial offering. Stay curious
and keep questioning everything.

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Thứ Sáu, 30 tháng 9, 2016

Breakthrough in Neuroscience: Stimulating Neurons Could Protect Against Brain Damage

Researchers have discovered a previously unknown mechanism that allows neural networks to protect against the spread of secondary brain damage as seen in TBI and ischemic stroke.



A breakthrough in understanding how brain damage spreads – and how it could potentially be limited – has been made through collaboration between neuroscientists and engineers at the Universities of Dundee and Strathclyde.

They have uncovered a previously unknown mechanism in the brain that allows networks of neurons to protect against the kind of spreading secondary damage seen in cases of strokes and traumatic brain injuries.

“If this network activity could be triggered clinically as soon as possible then major brain damage could be minimized and recovery periods shortened,” said Doctor Christopher Connolly, Reader in Neurobiology in the University of Dundee’s School of Medicine.



“Although this is basic laboratory research, it does now re-open the door to the possibility of stopping ongoing brain damage.

“Slow acting neuroprotection is well known but approaches to induce protection require at least 24 hours’ notice to be effective. This is of no practical use in a clinical emergency situation such as a stroke or traumatic brain injury, so current treatment options are limited to aiding the recovery processes.

“We have identified that neuronal networks react to an insult by sending rapid – in minutes – warning signals in an attempt to protect against the toxicity that causes brain damage. If that could be recruited clinically then it would give us a tool to deploy quickly in cases where brain damage was a risk.

“Where we can’t protect neurons quickly, we can recruit the help of surrounding neurons to do this for us. It is a case of `If you need a job done quickly, ask the expert’ and in this instance the experts are the neurons themselves.”



Laboratory-based modelling also showed that the rapid use of benzodiazepines (Valium) appeared to mimic the protection offered by the neuron networks.

“This is something we certainly need to test further but it does suggest the possibility of an effective and immediate pharmacological treatment for stroke,” said Doctor Connolly.

Image of the microfluidic device developed to determine activity-dependent spreading neurotoxic and neuroprotective signaling. Five parallel cell culture chambers recreate in vivo disease conditions. NeuroscienceNews.com image is adapted from the University of Strathclyde press release.

Doctor Connolly worked on the project with Doctor Michele Zagnoni, Senior Lecturer in Electronic and Electrical Engineering at the University of Strathclyde.

Doctor Zagnoni said, “Using microfluidic technology, we were able to produce in-vitro neuronal networks to investigate spreading toxicity in the brain, which is the cause of brain damage even after an initial trauma.

“Through this process we were able to demonstrate how the spread of this toxicity is driven. In doing that we also uncovered a previously unknown, fast acting, neuroprotective signaling mechanism.

“This mechanism utilizes the innate capacity of the surrounding neuronal networks (grown in the laboratory) to provide protection against the spreading toxicity. By stimulating that network, then theoretically we could limit the spread of brain damage. That requires further work, but it is an exciting and important possibility.”



The results of the research are published in the journal Scientific Reports.

The project examined the process known as acute secondary neuronal cell death, which is seen in neurodegenerative disease, cerebral ischemia (stroke) and traumatic brain injury (TBI) and drives spreading neurotoxicity into surrounding, undamaged, brain areas.

Source: University of Strathclyde.

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